Inflammation is classically regarded as a feature of innate immunity. It is characterized by redness, swelling at the site of injury and pain due to the influx of neutrophils, leukocytes and quickly followed by the influx of monocytes. Monocytes mature to inflammatory macrophages, proliferate and eventually alter the cellular function. The initiation and rapid onset of inflammation is mediated through receptors, unlike in adaptive immunity and involves 4 stages- a trigger by the stimulant (sterile/ infection), a radar that recognises the danger (receptors), signal transduction to the nucleus to initiate production of various mediators for combating the damage and clearance of the stimuli. The inflammatory response is the coordinate activation of signalling pathways that regulate inflammatory mediator levels in resident tissue cells and inflammatory cells recruited from the blood. Inflammation is a common pathogenesis of many chronic diseases, including cardiovascular and bowel diseases, diabetes, arthritis, and cancer (Lugrin et al., 2014).